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Published by Oxford University Press on behalf of the Guarantors of Brain. All rights reserved. ","subitem_rights_language":"en"}]},"item_1617186609386":{"attribute_name":"主題","attribute_value_mlt":[{"subitem_subject":"神経変性疾患","subitem_subject_language":"ja","subitem_subject_scheme":"Other"},{"subitem_subject":"neurodegenerative disease","subitem_subject_language":"en","subitem_subject_scheme":"Other"},{"subitem_subject":"prevention","subitem_subject_language":"en","subitem_subject_scheme":"Other"},{"subitem_subject":"oligomer","subitem_subject_language":"en","subitem_subject_scheme":"Other"},{"subitem_subject":"mitochondria","subitem_subject_language":"en","subitem_subject_scheme":"Other"},{"subitem_subject":"autophagy","subitem_subject_language":"en","subitem_subject_scheme":"Other"}]},"item_1617186626617":{"attribute_name":"内容記述","attribute_value_mlt":[{"subitem_description":"Amyloid-β, tau, and α-synuclein, or more specifically their soluble oligomers, are the aetiologic molecules in Alzheimer's disease, tauopathies, and α-synucleinopathies, respectively. These proteins have been shown to interact to accelerate each other's pathology. Clinical studies of amyloid-β-targeting therapies in Alzheimer's disease have revealed that the treatments after disease onset have little benefit on patient cognition....","subitem_description_language":"en","subitem_description_type":"Abstract"},{"subitem_description":"This study was supported by the Grants-in-Aid for Scientific Research from the Ministry of Education, Culture, Sports, Science and Technology of Japan (no. 23110514, 24659434, 25290018); by the Grants-in-Aid from the Ministry of Health, Labour, and Welfare, Japan; and in part by the Strategic Research Program for Brain Sciences (CREST), the Ministry of Education, Culture, Sports, Science and Technology of Japan.","subitem_description_language":"en","subitem_description_type":"Other"}]},"item_1617186643794":{"attribute_name":"出版者","attribute_value_mlt":[{"subitem_publisher":"Oxford University Press","subitem_publisher_language":"en"}]},"item_1617186783814":{"attribute_name":"識別子","attribute_value_mlt":[{"subitem_identifier_type":"DOI","subitem_identifier_uri":"https://doi.org/10.1093/brain/aww042"}]},"item_1617186920753":{"attribute_name":"収録物識別子","attribute_value_mlt":[{"subitem_source_identifier":"0006-8950","subitem_source_identifier_type":"ISSN"}]},"item_1617187056579":{"attribute_name":"書誌情報","attribute_value_mlt":[{"bibliographicIssueDates":{"bibliographicIssueDate":"2016-03-28","bibliographicIssueDateType":"Issued"},"bibliographicIssueNumber":"5","bibliographicPageEnd":"1586","bibliographicPageStart":"1568","bibliographicVolumeNumber":"139","bibliographic_titles":[{"bibliographic_title":"Brain","bibliographic_titleLang":"en"}]}]},"item_1617258105262":{"attribute_name":"資源タイプ","attribute_value_mlt":[{"resourcetype":"journal article","resourceuri":"http://purl.org/coar/resource_type/c_6501"}]},"item_1617265215918":{"attribute_name":"出版タイプ","attribute_value_mlt":[{"subitem_version_resource":"http://purl.org/coar/version/c_b1a7d7d4d402bcce","subitem_version_type":"AO"}]},"item_1617353299429":{"attribute_name":"関連情報","attribute_value_mlt":[{"subitem_relation_type":"isVersionOf","subitem_relation_type_id":{"subitem_relation_type_id_text":"https://doi.org/10.1093/brain/aww042","subitem_relation_type_select":"DOI"}}]},"item_1722232185104":{"attribute_name":"ファイル情報","attribute_type":"file","attribute_value_mlt":[{"accessrole":"open_date","date":[{"dateType":"Available","dateValue":"2017-12-19"}],"displaytype":"detail","filename":"00068950-139-5-1568.pdf","filesize":[{"value":"1.8 MB"}],"format":"application/pdf","mimetype":"application/pdf","url":{"label":"本文","objectType":"fulltext","url":"https://ocu-omu.repo.nii.ac.jp/record/2019690/files/00068950-139-5-1568.pdf"},"version_id":"905c4a54-9f63-4fc0-913e-a4d90351c63a"}]},"item_1722577087681":{"attribute_name":"記事種別等","attribute_value_mlt":[{"subitem_description":"Editor's Choice ","subitem_description_language":"en","subitem_description_type":"Other"}]},"item_1725590125188":{"attribute_name":"言語","attribute_value_mlt":[{"subitem_language":"eng"}]},"item_title":"Rifampicin is a candidate preventive medicine against amyloid-β and tau oligomers","item_type_id":"40002","owner":"7","path":["1721355153493"],"pubdate":{"attribute_name":"PubDate","attribute_value":"2017-12-19"},"publish_date":"2017-12-19","publish_status":"0","recid":"2019690","relation_version_is_last":true,"title":["Rifampicin is a candidate preventive medicine against amyloid-β and tau oligomers"],"weko_creator_id":"7","weko_shared_id":-1},"updated":"2024-10-31T00:59:26.956777+00:00"}